By Ronald K. Ross (auth.), Kenneth J. Pienta (eds.)
The box of genito-urinary oncology is quickly evolving at almost each point. major advances were made in our realizing of the molecular and mobile occasions which give a contribution to the iteration of GU malignancies. even as, related advances were made within the scientific enviornment that have stronger the prognosis and remedy of urologic cancers.
This quantity makes an attempt to summarize these advances which so much effect us as clinicians, and has been divided into 3 sections. part One, `Diagnostic advances: using molecular drugs within the analysis and analysis of GU malignancies', information how epidemiologic experiences and new molecular recommendations are impacting our skill to diagnose and deal with GU tumors. part , `Surgical and radiation advances', info the hot significant advances within the remedy of organ-confined cancers. part 3, `Medical advances', addresses significant matters within the remedy of metastatic disorder. This quantity will function a compendium of the advances, either on the simple technology and medical degrees, that are at present impacting training oncologists and urologists.
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Extra resources for Diagnosis and Treatment of Genitourinary Malignancies
27 Retinal hemangioblastomas These lesions have traditionally been called 'retinal angiomas' but are histologically identical of eNS hemangioblastomas . The tumors do not metastasize; however, they can cause glaucoma and blindness by virtue of unchecked growth, retinal detachment, or extravasation of serum from the vasculature. They are among the earliest manifestation of VHL and have been diagnosed in individuals ages 1 to 90, with an average age at diagnosis of 25 years [27,52]. Diagnosis of subtle, preangiomatous lesions may require indirect ophthalmoscopy with or without flourescein angiography and tonometry.
Point mutations as diagnostic markers of cancer Although some researchers claim that the detection of certain point mutations is diagnostic of cancer , others argue that the summation of numerous genetic alterations defines the cancer phenotype . Larger studies need to be done to determine the frequency with which people with point mutations in their p53 gene develop clinical tumors. The significance and timing of p53 mutations seems to be different for different cancers, being an early phenomenon in prostate cancer and a late phenomenon in colon cancer.
57. Walther MM, Reiter R, Keiser HR, et al. (submitted). Pheochromocytoma in von Hippel- Lindau disease: clinical and genetic characterization. 58. Richard S, Beigelman C, Duclos JM, et al. (1994). Pheochromocytoma as the first manifestation of von Hippel-Lindau disease. Surgery 116:1076. 59. Neumann HPH, Dinkel E, Brambs H, et al. (1991). Pancreatic lesions in the von HippelLindau syndrome. Gastroenterology 101:465. 60. Deboever G, Dequif P, Maertens J (1992). Common bile duct obstruction due to pancreatic involvement in the von Hippel-Lindau syndrome.
Diagnosis and Treatment of Genitourinary Malignancies by Ronald K. Ross (auth.), Kenneth J. Pienta (eds.)