Download PDF by Shweta S. Puntambekar, Jonathan M. Doose (auth.), Thomas E.: Central Nervous System Diseases and Inflammation

By Shweta S. Puntambekar, Jonathan M. Doose (auth.), Thomas E. Lane, Monica Carson, Conni Bergmann, Tony Wyss-Coray (eds.)

ISBN-10: 0387738932

ISBN-13: 9780387738932

ISBN-10: 0387738940

ISBN-13: 9780387738949

This quantity serves as a succinct and well-organized reference quantity keen on inflammatory CNS ailment to a large viewers. The textual content is created from 4 sections revolving round present components of curiosity within the fields of neuroimmunology, virology, pharmacology and affliction. Sections of this article specialise in a selected type of illnesses in addition to the pharmacological, virological, and immunological results of and at the illness.

Although many have lined this subject, few have supplied a quantity as concise and effective. each one disorder is damaged down on quite a few medical degrees with out entering into the heritage of the disorder. This strategy presents a succinct assessment of a selected topic encompassing interrelated issues bearing on neurology, immunology and disorder. the knowledge supplied in primary apprehensive approach ailments and irritation should be valuable for researchers, clinicians, in addition to a important source for college students drawn to the interesting area of neuroinflammation.

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2002). , 1997). Hypoxia also induces Ang-2 without changing the expression patterns of Ang-1 (Mandriota and Pepper, 1998). Therefore Ang-2 has a rather pro-angiogenic effect whereas Ang-1 and its source, the pericytes, act in a stabilizing way. Pericyte recruitment to sites of angiogenesis is another requirement for proper vascular development. This recruitment is driven by PDGF-&betabdot; and the corresponding receptor PDGFR-β. , 1997). Mice lacking PDGF-&betabdot; or its receptor develop identical phenotypes.

Lacor, P. , Lambert, M. , Finch, C. , Krafft, G. A. and Klein, W. L. (2003). Alzheimer’s disease-affected brain: presence of oligomeric A beta ligands (ADDLs) suggests a molecular basis for reversible memory loss. Proc Natl Acad Sci U S A, 100, 10417–10422. Gordon, M. , Holcomb, L. , Jantzen, P. , Boyett, K. , O’Callaghan, J. P. and Morgan, D. (2002). Time course of the development of Alzheimer-like pathology in the doubly transgenic PS1 + APP mouse. Exp Neurol, 173, 183–195. Griffin, W. , Sheng, J.

Am J Pathol, 139, 589–598. , Banati, R. , Masters, C. L. and Beyreuther, K. (1995). Extracellular matrix influences the biogenesis of amyloid precursor protein in microglial cells. J Biol Chem, 270, 7104–7110. , Oida, T. and Weiner, H. L. (2003). Microglia-mediated nitric oxide cytotoxicity of T cells following amyloid beta-peptide presentation to Th1 cells. J Immunol, 171, 2216–2224. Mook-Jung, I. and Saitoh, T. (1997). Amyloid precursor protein activates phosphotyrosine signaling pathway. Neurosci Lett, 235, 1–4.

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Central Nervous System Diseases and Inflammation by Shweta S. Puntambekar, Jonathan M. Doose (auth.), Thomas E. Lane, Monica Carson, Conni Bergmann, Tony Wyss-Coray (eds.)


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