Get Biopsy Pathology in Colorectal Disease, 2Ed PDF

By Talbot, Ian

ISBN-10: 0340759224

ISBN-13: 9780340759226

ISBN-10: 1444113569

ISBN-13: 9781444113563

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Example text

1984). for proximal inflammation when seen in contrast to the left side. 3) in each crypt is increased in inflammatory disease of long standing, particularly ulcerative colitis (Fig. , 1986a,b). It has been suggested by Lampert et al. 6). The clearest evidence of inflammation is an increase in cellularity of the lamina propria and loss of the above gradient. The excess cells may be neutrophil polymorphs, lymphocytes, plasma cells or eosinophils in varying proportions. It is an excess of neutrophil polymorphs which is the essential marker of active versus inactive chronic inflammation.

56 Granulation tissue from a patient with ulcerative colitis containing, beneath surface exudate, bizarre primitive cells of possible endothelial or fibroblastic lineage. 10). In non-malignant strictures fibres of the muscularis mucosae are often intermingled with fibrous tissue and this arrangement can replace the loose connective tissue of the submucosa. This is not a feature specific to any particular underlying disease, but does occur in Crohn’s disease and may occasionally be detected along the deep margin of well-taken biopsies.

6b). 32 Neutrophils. As well as being scattered throughout the lamina propria, neutrophil polymorphs are present within the epithelium of the surface and crypts. Rectal biopsy from a patient with infective colitis. 13). 4 Lymphocytes Since lymphocytes are numerous in the lamina propria of normal mucosa, an increase in numbers is difficult to assess and only a large increase is of diagnostic significance. Excluding lymphomas, such an excess is rarely homogeneous, and usually takes the form of hyperplasia of the normal rectal mucosal lymphoid follicles (Fig.

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Biopsy Pathology in Colorectal Disease, 2Ed by Talbot, Ian

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