By Professor Dr. Gianni Boris Bradač, Priv.-Doz. Dr. Roland Oberson (auth.)
In this age once we are witnessing a veritable explosion in new modalities in diagnos tic imaging we proceed to have a superb want for distinct experiences of the vascularity of the mind in sufferers who've all kinds of cerebral vascular illness. a lot of the knowledge of cerebral vascular occlusive lesions which we built within the final 20 years used to be in accordance with our skill to illustrate the vessels that have been affected. a lot experimental paintings in animals have been performed the place significant cerebral vessels have been obstructed and the results of those obstructions at the mind saw pathologically. notwithstanding, it used to be no longer until eventually cerebral angiography might be played with the element that turned attainable within the a long time of the '60 's and as a consequence that lets start to comprehend the connection of the obstructed vessels saw angiographically to the medical findings. moreover, a lot physiologic info was once got. for example, the idea that ofluxury perfusion that's used to explain non-nutritional movement throughout the tissues was once saw first angiographically even supposing the time period used to be no longer used till LASSEN defined it as a pathophysiological phenomenon saw in the course of cerebral blood movement reports with radioactive isotopes. the idea that of embolic occlusions of the cerebral vessels as opposed to thrombosis was once clarified and the relative frequency of thrombosis as opposed to embolism was once higher understood. the concept that of collateral stream of the mind via so-called meningeal end-to finish arterial anastomoses was once enormously higher understood while serial angiography in obstructive cerebral vascular sickness used to be conducted with expanding frequency.
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Extra info for Angiography and Computed Tomography in Cerebro-Arterial Occlusive Diseases
It is surprising how frequently other "secondary lesions" are found together with the clinically suspected lesion on the angiogram. A careful description of all lesions is mandatory since in some cases only the multiplicity of lesions gives a clue to the symptomatology of the patient (Figs. 17, 18, 22, 23). On the other hand, lesions may be present and sometimes may also be very extensive without corresponding clinical symptoms (Figs. 14, 15, 16,23). 5 Tortuosity In accordance with the description of WEIBEL and FIELDS (1965a, b, 1969) the elongation of the internal carotid artery is classified according to the following groups: Tortuosity: S- or C-shaped elongation Coiling: Exaggerated S-shaped or circular curve (Fig.
The left subclavian artery (SL) and the right subclavian artery (SR) also have a common origin at the aortic arch c d 27 a Fig. 2a and b. Normal carotid angiogram; arterial phase (same indications). a Lateral view; b Anterior-posterior view. 3a and b. Normal vertebral angiogram ; arterial phase (same indications). a Lateral view; b anterior-posterior view. The catheter was in the left vertebral artery (VL). Partial retrograde filling of the right vertebral artery (VR); bilateral symmetric filling of a large anterior inferior cerebellar artery (Aica).
1961) and 10% (CIOFFI et al. 8% (VANNIX et al. 1977). 5% kinking. It is not easy to state which role these changes play in the etiopathogenesis of an ischemic disease. , tortuosity, different positions of the neck and head, generalized atherosclerosis, and eventually anatomic variations that impede the formation of collateral flow. In some of these cases surgical correction for coiling and kinking may be indicated. In a few patients examined after TIA, a coiling or kinking of the internal carotid artery was the only suspected pathologic finding (Figs.
Angiography and Computed Tomography in Cerebro-Arterial Occlusive Diseases by Professor Dr. Gianni Boris Bradač, Priv.-Doz. Dr. Roland Oberson (auth.)